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polyclonal rabbit anti-acc1  (Millipore)


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    Structured Review

    Millipore polyclonal rabbit anti-acc1
    Polyclonal Rabbit Anti Acc1, supplied by Millipore, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/polyclonal+rabbit+anti-acc1/acc+1/pmc12180712-5-0-4
    Average 90 stars, based on 1 article reviews
    polyclonal rabbit anti-acc1 - by Bioz Stars, 2026-09
    90/100 stars

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    Related Articles

    other:

    Article Title: Deletion of AMP-activated protein kinase impairs metastasis and is rescued by ROS scavenging or ectopic CD36 expression
    Article Snippet: Polyclonal rabbit anti-ACC1 , Millipore , Cat. # ABS1068.

    Article Title: CD36 maintains lipid homeostasis via selective uptake of monounsaturated fatty acids during matrix detachment and tumor progression
    Article Snippet: Polyclonal rabbit anti-ACC1 , Millipore , Cat. #ABS1068.



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    Cell Signaling Technology Inc abs1068 polyclonal rabbit anti acc1 cell signaling technology cat
    CD36-deficiency reduces HFD-induced hepatic fatty acid synthesis, but aggravates MCD-induced oxidative stress. (A–C, E) Total RNA and proteins were extracted from a piece of liver. The proteins expression of nSREBP2, FASN, <t>ACC1,</t> CD36 (A) , PGC1α, CPT1α, PPARα, CD36 (Β) , SOD1 and SOD2 (E) were determined by Western blot. The mRNA expression of PGC1α and PPARα were determined qPCR (C) . (D) ROS in the liver was determined by Dihydrothidium (DHE) staining. *P < 0.05; ns, no significance (n = 6).
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    Image Search Results


    CD36-deficiency reduces HFD-induced hepatic fatty acid synthesis, but aggravates MCD-induced oxidative stress. (A–C, E) Total RNA and proteins were extracted from a piece of liver. The proteins expression of nSREBP2, FASN, ACC1, CD36 (A) , PGC1α, CPT1α, PPARα, CD36 (Β) , SOD1 and SOD2 (E) were determined by Western blot. The mRNA expression of PGC1α and PPARα were determined qPCR (C) . (D) ROS in the liver was determined by Dihydrothidium (DHE) staining. *P < 0.05; ns, no significance (n = 6).

    Journal: Frontiers in Pharmacology

    Article Title: Reversed role of CD36 deficiency in high-fat diet or methionine/choline-deficient diet-induced hepatic steatosis and steatohepatitis

    doi: 10.3389/fphar.2025.1522177

    Figure Lengend Snippet: CD36-deficiency reduces HFD-induced hepatic fatty acid synthesis, but aggravates MCD-induced oxidative stress. (A–C, E) Total RNA and proteins were extracted from a piece of liver. The proteins expression of nSREBP2, FASN, ACC1, CD36 (A) , PGC1α, CPT1α, PPARα, CD36 (Β) , SOD1 and SOD2 (E) were determined by Western blot. The mRNA expression of PGC1α and PPARα were determined qPCR (C) . (D) ROS in the liver was determined by Dihydrothidium (DHE) staining. *P < 0.05; ns, no significance (n = 6).

    Article Snippet: Rabbit anti-acetyl-CoA carboxylase alpha (ACC1) polyclonal antibody was purchased from Cell Signaling Technology (Danvers, MA, United States).

    Techniques: Expressing, Western Blot, Staining